Review



non-phosphorylation index stat3 antibody  (Cell Signaling Technology Inc)


Bioz Verified Symbol Cell Signaling Technology Inc is a verified supplier
Bioz Manufacturer Symbol Cell Signaling Technology Inc manufactures this product  
  • Logo
  • About
  • News
  • Press Release
  • Team
  • Advisors
  • Partners
  • Contact
  • Bioz Stars
  • Bioz vStars
    • 90
      Buy from Supplier

    Structured Review

    Cell Signaling Technology Inc non-phosphorylation index stat3 antibody
    MaR1 inhibits <t>NF-κB/STAT3</t> pathway activation in the kidney of sepsis mice. The expressions of <t>p-p65,</t> p65 (A) and p-Stat3, Stat3 (B) in renal tissues of mice in each group were analyzed by Western blot. The p-p65 protein levels normalized by p65 (C) , p-p65 protein levels normalized by β-actin (D) , p-Stat3 protein levels normalized by Stat3 (E) . Data are shown as mean ± SD, n = 9. ** P 0.01 vs. sham group; # P 0.05, ## P 0.01 vs. CLP group; && P 0.01 vs LD-MaR1 group. MaR1, Maresin 1; NF-κB, nuclear factor-kappa B; STAT3, signal transducer and activator of transcriptor 3; CLP, cecal ligation and puncture; LD-MaR1, MaR1 low-dose group.
    Non Phosphorylation Index Stat3 Antibody, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/non-phosphorylation index stat3 antibody/product/Cell Signaling Technology Inc
    Average 90 stars, based on 1 article reviews
    non-phosphorylation index stat3 antibody - by Bioz Stars, 2026-02
    90/100 stars

    Images

    1) Product Images from "Maresin 1 Mitigates Sepsis-Associated Acute Kidney Injury in Mice via Inhibition of the NF-κB/STAT3/MAPK Pathways"

    Article Title: Maresin 1 Mitigates Sepsis-Associated Acute Kidney Injury in Mice via Inhibition of the NF-κB/STAT3/MAPK Pathways

    Journal: Frontiers in Pharmacology

    doi: 10.3389/fphar.2019.01323

    MaR1 inhibits NF-κB/STAT3 pathway activation in the kidney of sepsis mice. The expressions of p-p65, p65 (A) and p-Stat3, Stat3 (B) in renal tissues of mice in each group were analyzed by Western blot. The p-p65 protein levels normalized by p65 (C) , p-p65 protein levels normalized by β-actin (D) , p-Stat3 protein levels normalized by Stat3 (E) . Data are shown as mean ± SD, n = 9. ** P 0.01 vs. sham group; # P 0.05, ## P 0.01 vs. CLP group; && P 0.01 vs LD-MaR1 group. MaR1, Maresin 1; NF-κB, nuclear factor-kappa B; STAT3, signal transducer and activator of transcriptor 3; CLP, cecal ligation and puncture; LD-MaR1, MaR1 low-dose group.
    Figure Legend Snippet: MaR1 inhibits NF-κB/STAT3 pathway activation in the kidney of sepsis mice. The expressions of p-p65, p65 (A) and p-Stat3, Stat3 (B) in renal tissues of mice in each group were analyzed by Western blot. The p-p65 protein levels normalized by p65 (C) , p-p65 protein levels normalized by β-actin (D) , p-Stat3 protein levels normalized by Stat3 (E) . Data are shown as mean ± SD, n = 9. ** P 0.01 vs. sham group; # P 0.05, ## P 0.01 vs. CLP group; && P 0.01 vs LD-MaR1 group. MaR1, Maresin 1; NF-κB, nuclear factor-kappa B; STAT3, signal transducer and activator of transcriptor 3; CLP, cecal ligation and puncture; LD-MaR1, MaR1 low-dose group.

    Techniques Used: Activation Assay, Western Blot, Ligation

    MaR1 inhibits p65 nuclear translocation in the kidney of sepsis mice. Representative immunofluorescence staining of kidney tissue at 24 h after CLP in each group of mice, and white arrow indicates p65 into the nucleus, magnification 1,000×. MaR1, Maresin 1; CLP, cecal ligation and puncture.
    Figure Legend Snippet: MaR1 inhibits p65 nuclear translocation in the kidney of sepsis mice. Representative immunofluorescence staining of kidney tissue at 24 h after CLP in each group of mice, and white arrow indicates p65 into the nucleus, magnification 1,000×. MaR1, Maresin 1; CLP, cecal ligation and puncture.

    Techniques Used: Translocation Assay, Immunofluorescence, Staining, Ligation

    MaR1 inhibits MAPK pathway activation and p65 nuclear translocation in the kidney of sepsis mice. The expressions of p-JNK, p-ERK, and p-p38 in renal tissues of mice in each group were analyzed by Western blot (A) . The p-JNK (B) , p-ERK (C) , and p-p38 (D) protein levels normalized by β-actin. (E) Nuclear translocation of p65 count/field. Data are shown as mean ± SD, n = 9. ** P 0.01 vs. sham group; ## P 0.01 vs. CLP group; && P 0.01 vs. LD-MaR1 group. MaR1, Maresin 1; MAPK, mitogen-activated protein kinase; CLP, cecal ligation and puncture; LD-MaR1, MaR1 low-dose group.
    Figure Legend Snippet: MaR1 inhibits MAPK pathway activation and p65 nuclear translocation in the kidney of sepsis mice. The expressions of p-JNK, p-ERK, and p-p38 in renal tissues of mice in each group were analyzed by Western blot (A) . The p-JNK (B) , p-ERK (C) , and p-p38 (D) protein levels normalized by β-actin. (E) Nuclear translocation of p65 count/field. Data are shown as mean ± SD, n = 9. ** P 0.01 vs. sham group; ## P 0.01 vs. CLP group; && P 0.01 vs. LD-MaR1 group. MaR1, Maresin 1; MAPK, mitogen-activated protein kinase; CLP, cecal ligation and puncture; LD-MaR1, MaR1 low-dose group.

    Techniques Used: Activation Assay, Translocation Assay, Western Blot, Ligation



    Similar Products

    non-phosphorylation index stat3 antibody  (Cell Signaling Technology Inc)
    90
    Cell Signaling Technology Inc non-phosphorylation index stat3 antibody
    MaR1 inhibits <t>NF-κB/STAT3</t> pathway activation in the kidney of sepsis mice. The expressions of <t>p-p65,</t> p65 (A) and p-Stat3, Stat3 (B) in renal tissues of mice in each group were analyzed by Western blot. The p-p65 protein levels normalized by p65 (C) , p-p65 protein levels normalized by β-actin (D) , p-Stat3 protein levels normalized by Stat3 (E) . Data are shown as mean ± SD, n = 9. ** P 0.01 vs. sham group; # P 0.05, ## P 0.01 vs. CLP group; && P 0.01 vs LD-MaR1 group. MaR1, Maresin 1; NF-κB, nuclear factor-kappa B; STAT3, signal transducer and activator of transcriptor 3; CLP, cecal ligation and puncture; LD-MaR1, MaR1 low-dose group.
    Non Phosphorylation Index Stat3 Antibody, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/non-phosphorylation index stat3 antibody/product/Cell Signaling Technology Inc
    Average 90 stars, based on 1 article reviews
    non-phosphorylation index stat3 antibody - by Bioz Stars, 2026-02
    90/100 stars
    		  Buy from Supplier

    Image Search Results


    MaR1 inhibits NF-κB/STAT3 pathway activation in the kidney of sepsis mice. The expressions of p-p65, p65 (A) and p-Stat3, Stat3 (B) in renal tissues of mice in each group were analyzed by Western blot. The p-p65 protein levels normalized by p65 (C) , p-p65 protein levels normalized by β-actin (D) , p-Stat3 protein levels normalized by Stat3 (E) . Data are shown as mean ± SD, n = 9. ** P 0.01 vs. sham group; # P 0.05, ## P 0.01 vs. CLP group; && P 0.01 vs LD-MaR1 group. MaR1, Maresin 1; NF-κB, nuclear factor-kappa B; STAT3, signal transducer and activator of transcriptor 3; CLP, cecal ligation and puncture; LD-MaR1, MaR1 low-dose group.

    Journal: Frontiers in Pharmacology

    Article Title: Maresin 1 Mitigates Sepsis-Associated Acute Kidney Injury in Mice via Inhibition of the NF-κB/STAT3/MAPK Pathways

    doi: 10.3389/fphar.2019.01323

    Figure Lengend Snippet: MaR1 inhibits NF-κB/STAT3 pathway activation in the kidney of sepsis mice. The expressions of p-p65, p65 (A) and p-Stat3, Stat3 (B) in renal tissues of mice in each group were analyzed by Western blot. The p-p65 protein levels normalized by p65 (C) , p-p65 protein levels normalized by β-actin (D) , p-Stat3 protein levels normalized by Stat3 (E) . Data are shown as mean ± SD, n = 9. ** P 0.01 vs. sham group; # P 0.05, ## P 0.01 vs. CLP group; && P 0.01 vs LD-MaR1 group. MaR1, Maresin 1; NF-κB, nuclear factor-kappa B; STAT3, signal transducer and activator of transcriptor 3; CLP, cecal ligation and puncture; LD-MaR1, MaR1 low-dose group.

    Article Snippet: The membranes were incubated with the following primary antibody at 4°C overnight: phosphorylation index (p-p65, p-Stat3, p-JNK, p-ERK, p-p38), non-phosphorylation index (p65, Stat3), and β-actin (Cell Signaling Technology, Danvers, MA, USA) at 1:1,000.

    Techniques: Activation Assay, Western Blot, Ligation

    MaR1 inhibits p65 nuclear translocation in the kidney of sepsis mice. Representative immunofluorescence staining of kidney tissue at 24 h after CLP in each group of mice, and white arrow indicates p65 into the nucleus, magnification 1,000×. MaR1, Maresin 1; CLP, cecal ligation and puncture.

    Journal: Frontiers in Pharmacology

    Article Title: Maresin 1 Mitigates Sepsis-Associated Acute Kidney Injury in Mice via Inhibition of the NF-κB/STAT3/MAPK Pathways

    doi: 10.3389/fphar.2019.01323

    Figure Lengend Snippet: MaR1 inhibits p65 nuclear translocation in the kidney of sepsis mice. Representative immunofluorescence staining of kidney tissue at 24 h after CLP in each group of mice, and white arrow indicates p65 into the nucleus, magnification 1,000×. MaR1, Maresin 1; CLP, cecal ligation and puncture.

    Article Snippet: The membranes were incubated with the following primary antibody at 4°C overnight: phosphorylation index (p-p65, p-Stat3, p-JNK, p-ERK, p-p38), non-phosphorylation index (p65, Stat3), and β-actin (Cell Signaling Technology, Danvers, MA, USA) at 1:1,000.

    Techniques: Translocation Assay, Immunofluorescence, Staining, Ligation

    MaR1 inhibits MAPK pathway activation and p65 nuclear translocation in the kidney of sepsis mice. The expressions of p-JNK, p-ERK, and p-p38 in renal tissues of mice in each group were analyzed by Western blot (A) . The p-JNK (B) , p-ERK (C) , and p-p38 (D) protein levels normalized by β-actin. (E) Nuclear translocation of p65 count/field. Data are shown as mean ± SD, n = 9. ** P 0.01 vs. sham group; ## P 0.01 vs. CLP group; && P 0.01 vs. LD-MaR1 group. MaR1, Maresin 1; MAPK, mitogen-activated protein kinase; CLP, cecal ligation and puncture; LD-MaR1, MaR1 low-dose group.

    Journal: Frontiers in Pharmacology

    Article Title: Maresin 1 Mitigates Sepsis-Associated Acute Kidney Injury in Mice via Inhibition of the NF-κB/STAT3/MAPK Pathways

    doi: 10.3389/fphar.2019.01323

    Figure Lengend Snippet: MaR1 inhibits MAPK pathway activation and p65 nuclear translocation in the kidney of sepsis mice. The expressions of p-JNK, p-ERK, and p-p38 in renal tissues of mice in each group were analyzed by Western blot (A) . The p-JNK (B) , p-ERK (C) , and p-p38 (D) protein levels normalized by β-actin. (E) Nuclear translocation of p65 count/field. Data are shown as mean ± SD, n = 9. ** P 0.01 vs. sham group; ## P 0.01 vs. CLP group; && P 0.01 vs. LD-MaR1 group. MaR1, Maresin 1; MAPK, mitogen-activated protein kinase; CLP, cecal ligation and puncture; LD-MaR1, MaR1 low-dose group.

    Article Snippet: The membranes were incubated with the following primary antibody at 4°C overnight: phosphorylation index (p-p65, p-Stat3, p-JNK, p-ERK, p-p38), non-phosphorylation index (p65, Stat3), and β-actin (Cell Signaling Technology, Danvers, MA, USA) at 1:1,000.

    Techniques: Activation Assay, Translocation Assay, Western Blot, Ligation